Research & Education

IASLC Issues New Statement on Tobacco Control and Smoking Cessation

DENVER, Colo. — The International Association for the Study of Lung Cancer (IASLC) today issued a new statement on Tobacco Control and Smoking Cessation at the 16th World Conference on Lung Cancer (WCLC) in Denver. The statement calls for higher taxes on tobacco products, comprehensive advertising and promotion bans of all tobacco products and product regulation including pack warnings.

IASLC to hold Third Annual CT Screening Workshop

DENVER, Colo. – The International Association for the Study of Lung Cancer’s (IASLC) Strategic Screening Advisory Committee will hold the Third CT Screening Workshop on Saturday, Sept. 5 at 9 a.m. at the Colorado Convention Center, Rooms 708+710+712, prior to the official opening of the 16th Annual World Conference on Lung Cancer (WCLC). International leaders in computed tomography (CT) screening from across the world, including Japan, Australia, Canada, Europe and the U.S., are set to participate.

Dr. Fred R. Hirsch Addresses Unmet Needs in Lung Cancer Treatment

Speaking in an interview with Oncology Nursing News, Dr. Fred R. Hirsch, CEO of IASLC, Professor of Medicine and Pathology at the University of Colorado, discusses the importance of combination therapies in the fight against lung cancer.

IASLC Lung Cancer Staging Project - Proposals

IASLC Lung Cancer Staging Project: Proposals for the Revisions of the T Descriptors in the Forthcoming Eighth Edition of the TNM Classification for Lung Cancer

World No Tobacco Day - May 31, 2015 - Stop illicit trade of tobacco products.

One in every 10 cigarettes, and many other tobacco products, consumed worldwide are illegal, making the illicit trade of tobacco products a major global concern from many perspectives, including health, legal, economic, governance and corruption.

Driver of non-small cell lung cancer, FGFR1, also in 23 percent of small cell lung cancer

The study identifies a subset of patients with small cell lung cancer with potentially over activated FGFR1 pathways as evidenced by FGFR1 gene amplification, increased FGFR1 mRNA levels, and high protein expression.